Acute renal failure, also termed acute kidney injury, is a sudden decline in kidney function occurring over hours to days, characterised by the rapid accumulation of nitrogenous waste, disturbance of fluid and electrolyte balance, and often reduced urine output. It is conventionally categorised by mechanism as prere…
Acute renal failure, also termed acute kidney injury, is a sudden decline in kidney function occurring over hours to days, characterised by the rapid accumulation of nitrogenous waste, disturbance of fluid and electrolyte balance, and often reduced urine output. It is conventionally categorised by mechanism as prerenal, arising from impaired renal perfusion; intrinsic, from direct damage to the renal parenchyma; or postrenal, from urinary tract obstruction, with causes including infection, nephrotoxic exposure, ischaemia, and structural lesions. Prompt recognition and treatment are essential, since timely correction of reversible factors can limit lasting renal damage and improve outcomes. Research collected here reflects these dimensions, including retroperitoneal fibrosis as a cause of acute renal failure, the nephrotoxicity of iodinated radiographic contrast agents, and severe diarrhoea as a precipitant of morbidity and mortality in pediatric patients. Further contributions examine polycystic kidney disease, nephrotoxicity models in rats, rhabdomyolysis in an HIV-infected patient, single-nucleotide polymorphism profiling in acute renal rejection, and acute urinary retention. Together these peer-reviewed studies span the causes, mechanisms, and management of acute kidney injury, including nephrotoxic, obstructive, and systemic contributors to sudden renal dysfunction.
12 peer-reviewed articles, ranked by relevance. Each links to its DOI.
The 12 articles above have been cited 29 times in the scholarly literature. Citation data via OpenAlex and Crossref, updated Jun 2026.
A sample of recent works citing this journal's research on Acute Renal Failure, linking to each citing work.